61 research outputs found
A preliminary study of micro-gestures:dataset collection and analysis with multi-modal dynamic networks
Abstract. Micro-gestures (MG) are gestures that people performed spontaneously during communication situations. A preliminary exploration of Micro-Gesture is made in this thesis. By collecting recorded sequences of body gestures in a spontaneous state during games, a MG dataset is built through Kinect V2. A novel term âmicro-gestureâ is proposed by analyzing the properties of MG dataset. Implementations of two sets of neural network architectures are achieved for micro-gestures segmentation and recognition task, which are the DBN-HMM model and the 3DCNN-HMM model for skeleton data and RGB-D data respectively. We also explore a method for extracting neutral states used in the HMM structure by detecting the activity level of the gesture sequences. The method is simple to derive and implement, and proved to be effective. The DBN-HMM and 3DCNN-HMM architectures are evaluated on MG dataset and optimized for the properties of micro-gestures. Experimental results show that we are able to achieve micro-gesture segmentation and recognition with satisfied accuracy with these two models. The work we have done about the micro-gestures in this thesis also explores a new research path for gesture recognition. Therefore, we believe that our work could be widely used as a baseline for future research on micro-gestures
Fashion, Cooperation, and Social Interactions
Fashion plays such a crucial rule in the evolution of culture and society
that it is regarded as a second nature to the human being. Also, its impact on
economy is quite nontrivial. On what is fashionable, interestingly, there are
two viewpoints that are both extremely widespread but almost opposite:
conformists think that what is popular is fashionable, while rebels believe
that being different is the essence. Fashion color is fashionable in the first
sense, and Lady Gaga in the second. We investigate a model where the population
consists of the afore-mentioned two groups of people that are located on social
networks (a spatial cellular automata network and small-world networks). This
model captures two fundamental kinds of social interactions (coordination and
anti-coordination) simultaneously, and also has its own interest to game
theory: it is a hybrid model of pure competition and pure cooperation. This is
true because when a conformist meets a rebel, they play the zero sum matching
pennies game, which is pure competition. When two conformists (rebels) meet,
they play the (anti-) coordination game, which is pure cooperation. Simulation
shows that simple social interactions greatly promote cooperation: in most
cases people can reach an extraordinarily high level of cooperation, through a
selfish, myopic, naive, and local interacting dynamic (the best response
dynamic). We find that degree of synchronization also plays a critical role,
but mostly on the negative side. Four indices, namely cooperation degree,
average satisfaction degree, equilibrium ratio and complete ratio, are defined
and applied to measure people's cooperation levels from various angles. Phase
transition, as well as emergence of many interesting geographic patterns in the
cellular automata network, is also observed.Comment: 21 pages, 12 figure
Slit2âRobo4 signalling promotes vascular stability by blocking Arf6 activity
SlitâRoundabout (Robo) signalling has a well-understood role in axon guidance1â5. Unlike in the nervous system, however, Slitdependent activation of an endothelial-specific Robo, Robo4, does not initiate a guidance program. Instead, Robo4 maintains the barrier function of the mature vascular network by inhibiting neovascular tuft formation and endothelial hyperpermeability induced by pro-angiogenic factors 6. In this study, we used cell biological and biochemical techniques to elucidate the molecular mechanism underlying the maintenance of vascular stability by Robo4. Here, we demonstrate that Robo4 mediates Slit2-dependent suppression of cellular protrusive activity through direct interaction with the intracellular adaptor protein paxillin and its paralogue, Hic-5. Formation of a Robo4âpaxillin complex at the cell surface blocks activation of the small GTPase Arf6 and, consequently, Rac by recruitment of Arf-GAPs (ADP-ribosylation factor- directed GTPase-activating proteins) such as GIT1. Consistent with these in vitro studies, inhibition of Arf6 activity in vivo phenocopies Robo4 activation by reducing pathologic angiogenesis in choroidal and retinal vascular disease and VEGF-165 (vascular endothelial growth factor-165)-induced retinal hyperpermeability. These data reveal that a Slit2âRobo4âpaxillinâGIT1 network inhibits the cellular protrusive activity underlying neovascularization and vascular leak, and identify a new therapeutic target for ameliorating diseases involving the vascular system
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